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Environmental Health Research

Household Air Pollution Study

Indoor exposure to PM2.5, black carbon, and NO2 in New Orleans homes, and its effect on cardiovascular health.

Led by Dr. Felicia Rabito

The air inside your home.

The Household Air Pollution Study (HAPS) at Tulane University's Weatherhead School of Public Health measured indoor concentrations of PM2.5, black carbon, and nitrogen dioxide in homes across New Orleans.

Dr. Felicia Rabito's team placed monitors in participant homes to record exposure continuously, then compared pollutant levels with each participant's ambulatory blood pressure, respiratory function tests, and inflammatory biomarkers.

New Orleans is a useful place to study this. The housing is old, many homes cook with gas, the humid climate shapes how people ventilate, and several communities already carry a higher burden of cardiovascular disease.

Role

Research Assistant

I deployed and retrieved the PM2.5 and NO2 monitors in participant homes, coordinated with participants, followed the study's quality assurance protocols, and helped turn raw sensor output into datasets ready for analysis.

The work taught me how environmental measurements get tied to health outcomes in a real study.

Study Design

  • •Environmental monitoring: continuous PM2.5, black carbon, and NO2 sensors deployed in homes
  • •Biologic assessment: ambulatory blood pressure, respiratory function, inflammatory markers
  • •Population: New Orleans residents in neighborhoods with elevated environmental health risks
  • •Analysis: quartile-based exposure-response modeling for cardiovascular endpoints
Pollutant Profiles

Pollution sources in the home.

Gas appliances, cooking, candles, tobacco and traffic outside the door each add to the air inside a home. Filter by pollutant to see where it comes from and which instrument measured it.

Where indoor pollution comes from, and how the study measured it

Schematic section of a New Orleans shotgun house. Select a source or monitor for details.

  • PM2.5
  • Black carbon
  • NO₂ (gas)
  • Outdoor air seeping in

Illustrative schematic: source and monitor positions are representative, not a specific study home.

PM2.5

Fine particulate matter smaller than 2.5 micrometers in diameter. The particles reach deep into the lung alveoli and can enter the bloodstream, causing inflammation throughout the body. Indoor sources include cooking, candles, incense, and tobacco smoke.1

Sources
Gas space heater, Tobacco smoke, Candles & incense, Gas stove & cooking
Measured by
pDR-1500

WHO guideline (2021)4

5µg/m³

annual mean

15µg/m³

24-hour mean

Black carbon

A component of soot from incomplete combustion of fossil fuels, biomass, and cooking fuels. In the study, each 1 µg/m³ increase in residential black carbon was associated with a 7.55 mmHg increase in systolic blood pressure.2

Sources
Traffic exhaust, Tobacco smoke
Measured by
MicroAeth AE51

WHO guideline (2021)4

No guideline value. Evidence was judged insufficient to set one; WHO recommends measuring and reducing it.

NO₂

Indoors, nitrogen dioxide comes mostly from gas stoves and space heaters. NO₂ irritates the airways, worsens asthma, and contributes to chronic respiratory disease. Poorly ventilated homes are hit hardest.3

Sources
Traffic exhaust, Gas space heater, Gas stove & cooking
Measured by
Ogawa Samplers

WHO guideline (2021)4

10µg/m³

annual mean

25µg/m³

24-hour mean

WHO guideline values: WHO global air quality guidelines (2021), Table 0.1. 24-hour values are the 99th percentile, i.e. 3–4 exceedance days per year; the guidelines apply to indoor as well as outdoor air. For black carbon, WHO found the evidence insufficient to set a guideline level and issues good-practice statements instead.4

Published Finding

The evidence.

Per 1 µg/m³ of residential black carbon

+7.55mmHg

Systolic blood pressure increase

Each 1 µg/m³ increase in residential black carbon was associated with a 7.55 mmHg rise in systolic blood pressure.2 Across a whole population, a shift like this matters, especially in communities that already have high rates of cardiovascular disease.

What 2 mmHg means at population scale

  • ≈10%higher stroke mortality
  • ≈7%higher ischemic heart disease mortality

A 2 mmHg population-level increase in systolic BP is associated with a 7% increase in ischemic heart disease mortality and a 10% increase in stroke mortality (Lewington et al., Lancet 2002; meta-analysis of one million adults in 61 prospective studies).5 These are population associations, not deaths estimated from this study.

Methodology

Measurement instruments.

In home

pDR-1500

Real-time PM2.5

In home

MicroAeth AE51

Black carbon

In home

Ogawa Samplers

Passive NO₂

Worn by participant

Ambulatory BP Monitor

24-hr blood pressure

Sources

  1. 1Indoor Particulate Matter · U.S. Environmental Protection AgencyIndoor PM sources (cooking, candles, unvented space heaters, smoking) and outdoor particles that migrate indoors; small particles get deep into the lungs and some reach the bloodstream.
  2. 2The association between short-term residential black carbon concentration on blood pressure in a general population sample · Rabito FA, et al., Indoor Air, 2021+7.55 mmHg systolic blood pressure per 1 µg/m³ residential black carbon (P = .02); black carbon comes from incomplete combustion and is tied to traffic and cooking.
  3. 3Nitrogen Dioxide's Impact on Indoor Air Quality · U.S. Environmental Protection AgencyIndoor NO₂ from gas stoves and unvented heaters; respiratory irritation and asthma effects; venting reduces exposure.
  4. 4WHO global air quality guidelines: particulate matter (PM2.5 and PM10), ozone, nitrogen dioxide, sulfur dioxide and carbon monoxide · World Health Organization, Sept 22, 2021Table 0.1 guideline levels (PM2.5 5/15 µg/m³, NO₂ 10/25 µg/m³); good-practice statements only for black carbon.
  5. 5Age-specific relevance of usual blood pressure to vascular mortality: a meta-analysis of individual data for one million adults in 61 prospective studies · Lewington S, et al. (Prospective Studies Collaboration), The Lancet, Dec 14, 20022 mmHg lower usual systolic BP ≈ 7% lower IHD mortality and ≈ 10% lower stroke mortality.